Brytalearn.How things workFind something
Back to the experimentTHE EVIDENCE BEHIND THE EXPERIENCE

Two processes, one changing day: sources & model

Change a fictional sleep history, catch the moment two curves meet, and separate a local clock change from a biological clock. Then follow light information from eye to brain.

Scientific review · independent subject review pending

The source and model records are available for inspection. No external scientific reviewer has signed off yet.

sleep-1 · content 1 · setup format 1

What supports the explanation?

Professional brain and eye reference illustrations

Unmodified PNGs by Servier Medical Art, CC BY 4.0. Separate schematic timing callouts are authored by Brytalearn; no exact SCN segmentation is claimed.

Servier Medical Art · Brain

Eye cutaway and source image reuse terms

Anterior at right, optic-nerve exit at left; the retinal lining is distinct from the orange vitreous-body context. CC BY 4.0 with attribution.

Servier Medical Art · Eye structure

Circadian patterns, environmental cues and coordinated tissue clocks

Institutional fact sheet. Its fruit-fly molecular example is not presented as a complete human clock.

NIH NIGMS · Circadian Rhythms

Wake history, sleep need and light-related timing

Institutional sleep/wake-cycle explanation. General physiology is not an individual calibration.

NIH NHLBI · Your Sleep/Wake Cycle

Original two-process framework

Original publication record. The accessible record verifies its 1982 origin; numerical parameters here are transcribed from the later explicit mathematical presentation.

Borbély · Human Neurobiology, 1982

Wake/sleep-dependent pressure with circadian thresholds

Original 1984 model abstract. Pressure need not be identified with one neurochemical substance.

Daan, Beersma & Borbély · 1984

Exact equations and parameter set for this implementation

The two-process model section, equations 1–4 and Figure 1b. Other models and parameter sets in the paper are not mixed into this engine.

Skeldon, Dijk & Derks · PLOS ONE, 2014

Parameter estimates depend on individual and modeling choices

Original analysis in eight healthy young men, with EEG slow-wave activity and normalization choices. These results do not define defaults for all ages.

Rusterholz, Dürr & Achermann · Sleep, 2010

Direction of light-related phase resetting depends on biological timing

Original study of 21 entrained participants under a 6.7-hour laboratory light protocol, using melatonin phase. Its protocol is not a household-lamp recommendation.

Khalsa et al. · Journal of Physiology, 2003

Photosensitive retinal ganglion cells project to clock-related circuitry

Original rat/mouse melanopsin-cell study. Supports a neural retinal-to-SCN route, not direct light transmission through brain tissue.

Hattar et al. · Science, 2002

REM and NREM stages are distinct from the binary model

Institutional explanation of sleep stages. No stage sequence or EEG signal is generated by the two-process model.

NIH NHLBI · Sleep Phases and Stages

Prescribed circadian waveforms omit light-driven oscillator dynamics

Section 2 distinguishes this limitation. Optimization schedules from the paper are not implemented.

Yin, Julius & Wen · PLOS ONE, 2021

Pineal melatonin production is distinct from SCN coordination

Brain-structure descriptions, pineal-gland item. The added output callout abbreviates intervening neural signals.

NIH NINDS · Understanding Sleep

What this model assumes

  1. A mathematical model with one published parameter set and a specified initial condition. It is not fitted to a child, adult or patient.
  2. S is a dimensionless state. No alertness score, diagnostic result, driving-safety judgment, exam-performance prediction or personal sleep recommendation is calculated.
  3. Prescribed blocks mean fictional modeled sleep or wake, not time in bed and not a guarantee that a person can fall asleep on command.
  4. The fixed circadian wave has no light-dose input or adaptation dynamics. Clock-label changes, assumed phase changes and elapsed time remain separate operations.
  5. The added daytime sleep block increases total modeled sleep. The separate order experiment holds total durations equal. These answer different comparison questions.
  6. The model does not simulate sleep stages, melatonin concentrations, caffeine, medication, long-term sleep-restriction effects or individual sleep requirements.
  7. The at-home activity observes an ordinary routine or uses supplied fictional cards. It asks for no altered sleep, alarm, caffeine, medication or light behavior.
  8. The declared mathematical model: We use the two-process presentation in Skeldon, Dijk & Derks (2014), Figure 1b. Wake: dS/dt = (1 − S)/18.2 h. Sleep: dS/dt = −S/4.2 h. C = sin[2π(t − α)/24 h]. The boundaries are U = 0.60 + 0.10C and L = 0.17 + 0.10C. S and the thresholds are dimensionless. The reset S = 0.17, awake at t = 0, is an authored initial condition.
  9. What the computation does: Each branch uses its exact exponential solution. A bracketed first-crossing search switches states without resetting pressure. The plot and downloaded table sample that same trajectory. Removing circadian modulation is an intervention on this model’s structure; it is not a simulated treatment or brain lesion.
  10. A fixed wave does not model light adaptation: The quantitative circadian signal is prescribed. It has no light-response or entrainment equation. An assumed phase shift restarts a comparison; it is not the calculated result of turning on a lamp. The anatomy and light-experiment views explain separate biological evidence.
  11. Pressure, measurements and experience differ: EEG slow-wave activity has been used to estimate homeostatic dynamics. Parameters can vary with individuals, normalization and modeling choices. Our S is not measured EEG voltage, blood adenosine concentration, subjective sleepiness, accumulated brain damage or a health score.
  12. Sleep has stages this model does not generate: Human sleep includes REM and three NREM stages. A binary wake/sleep trace does not tell us those stages, dream content, learning performance or a person’s exact sleep requirement. Those questions need other observations and models.
  13. Light timing can change shift direction: Khalsa et al. studied a laboratory protocol with 6.7 hours of bright-light exposure and melatonin-phase measurements. Exposure centered before the critical phase produced delays; after it, advances. That biological phase convention is distinct from the arbitrary zero of our sine model. It is not a household-light schedule or a rule tied to one civil-clock hour.
  14. Anatomy and hormonal output are separate: The SCN region helps coordinate timing. Clock-related signaling also influences the pineal gland, which produces melatonin. The SCN is not itself the melatonin-producing gland. Added anatomical callouts are magnified teaching locations, not measured segmented nuclei.

What has been checked

Analytical reference cases, conservation or transition invariants, finite drawing commands, bounded setup parsing, discovery and route integrity are checked automatically. These checks do not establish anatomical fidelity, learner outcomes or browser/device compatibility. Independent subject review, learner trials, comprehensive accessibility review and browser video encoding checks remain pending.

Each source supports the associated claim. Sources do not certify this implementation or its visuals.

About the cover illustration

Original plot of the declared two-process sleep model. Curves are calculations, not physiological measurements from a person. Model parameters and independently verified event times are sourced in the lesson. The separate eye and brain references are credited to Servier Medical Art, CC BY 4.0.

Our review process